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William G. Blackard
Researcher at VCU Medical Center
Publications - 90
Citations - 5721
William G. Blackard is an academic researcher from VCU Medical Center. The author has contributed to research in topics: Insulin & Diabetes mellitus. The author has an hindex of 39, co-authored 90 publications receiving 5628 citations. Previous affiliations of William G. Blackard include Virginia Commonwealth University & Tulane University.
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Journal ArticleDOI
A direct effect of hyperinsulinemia on serum sex hormone-binding globulin levels in obese women with the polycystic ovary syndrome.
John E. Nestler,Linda P. Powers,Dennis W. Matt,Kenneth A. Steingold,Stephen R. Plymate,Roger S. Rittmaster,John N. Clore,William G. Blackard +7 more
TL;DR: It is suggested that hyperinsulinemia directly reduces serum SHBG levels in obese women with the polycystic ovary syndrome independently of any effect on serum sex steroids.
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Dehydroepiandrosterone Reduces Serum Low Density Lipoprotein Levels and Body Fat but Does not Alter Insulin Sensitivity in Normal Men
TL;DR: It is suggested that in normal men DHEA administration reduces body fat, increases muscle mass, and reduces serum low density lipoprotein cholesterol levels.
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Suppression of serum insulin by diazoxide reduces serum testosterone levels in obese women with polycystic ovary syndrome.
John E. Nestler,Cornelius Barlascini,Dennis W. Matt,Kenneth A. Steingold,Stephen R. Plymate,John N. Clore,William G. Blackard +6 more
TL;DR: The results suggest that hyperinsulinemia in obese women with polycystic ovary syndrome may directly increase serum testosterone levels.
Journal ArticleDOI
Portal and Peripheral Vein Immunoreactive Insulin Concentrations Before and After Glucose Infusion
TL;DR: A significant positive correlation between portal vein and peripheral vein IRI responses to glucose was noted and the absolute amount of “big” insulin in the portal vein was similar during the first phase and the early part of the second phase of insulin secretion.
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Adrenergic receptor control mechanism for growth hormone secretion
TL;DR: A stimulatory effect of alpha receptors and a possible inhibitory effect of beta receptors on growth hormone secretion are demonstrated and more prolonged hypoglycemia and lower plasma free fatty acid values may have been a factor in the greater HGH concentrations observed during beta blockade.