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William J. Bremner
Researcher at Veterans Health Administration
Publications - 26
Citations - 1291
William J. Bremner is an academic researcher from Veterans Health Administration. The author has contributed to research in topics: Testosterone & Gonadotropin. The author has an hindex of 16, co-authored 26 publications receiving 1261 citations. Previous affiliations of William J. Bremner include University of Washington.
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Metabolic and behavioral effects of high-dose, exogenous testosterone in healthy men.
TL;DR: There was a trend toward increased arousal and spontaneous erections during T administration, but this did not reach statistical significance, and little change was found in self-reported sexual and aggressive behaviors during the study.
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Effects of endogenous testosterone and estradiol on sexual behavior in normal young men.
TL;DR: In a randomized, double-blind study, a GnRH antagonist is used to induce acute, profound, reversible gonadal steroid deficiency in 9 normal men for 6 weeks and the role of endogenous testosterone or estradiol in the regulation of behavior in healthy, eugonadal men is studied.
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Physiologic Testosterone Levels in Normal Men Suppress High-Density Lipoprotein Cholesterol Levels
TL;DR: The results imply that androgen levels in the normal adult male range have a suppressive effect on HDL cholesterol concentration and may contribute to the increased risk for coronary artery disease in men.
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Physiological Levels of Estradiol Stimulate Plasma High Density Lipoprotein2 Cholesterol Levels in Normal Men
TL;DR: In men, physiological levels of E2 are important in maintaining plasma levels of HDL cholesterol, especially the HDL2 fraction, and observations suggest that estrogen, in the amount normally produced in men, may offer some degree of protection against cardiovascular disease in males, as they do in women.
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Comparison of a gonadotropin releasing-hormone antagonist plus testosterone (T) versus T alone as potential male contraceptive regimens.
Carrie J. Bagatell,Alvin M. Matsumoto,Richard B. Christensen,Jean E. Rivier,William J. Bremner +4 more
TL;DR: It is concluded that with the dosages of Nal-Glu and T used in this study, the addition of GnRH antagonist to a high-dose T regimen does not increase the ability of T to suppress spermatogenesis in healthy men.