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Yannick Chaval

Researcher at University of Toulouse

Publications -  65
Citations -  2076

Yannick Chaval is an academic researcher from University of Toulouse. The author has contributed to research in topics: Population & Hantavirus. The author has an hindex of 25, co-authored 62 publications receiving 1764 citations. Previous affiliations of Yannick Chaval include Centre national de la recherche scientifique & SupAgro.

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Revisiting the taxonomy of the Rattini tribe: a phylogeny-based delimitation of species boundaries

TL;DR: This study explores and highlights the limits of the current taxonomy of the Rattini tribe, one of the most difficult groups of mammals, and lays the foundations to better investigate rodent-born diseases in South East Asia.
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Epidemiology of Leptospira Transmitted by Rodents in Southeast Asia

TL;DR: Strain typing confirmed rodents as reservoirs for human leptospirosis, and differences in habitat requirements for L. interrogans and L. borgpetersenii supported differential transmission modes.
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Migration and recovery of the genetic diversity during the increasing density phase in cyclic vole populations.

TL;DR: It is provided that in cyclic populations of the fossorial water voles, the relative influence of drift operating during low density populations and migration occurring principally while population size increases interacts closely to maintain high genetic diversity.
Book

Protocols for field and laboratory rodent studies

TL;DR: Protocols for field and laboratory rodent studies present the best practices for the studies of rodents and rodent-borne pathogens and parasites from the field to the laboratory.
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Stress and demographic decline: a potential effect mediated by impairment of reproduction and immune function in cyclic vole populations.

TL;DR: A significant negative correlation was observed between concentrations of FCMs and both immunocompetence and body condition during population decline, which might reflect an impairment of the female reproductive capability in high densities and accelerated senescence affecting immune function during decline, both arising from chronic stress.