Yoshimasa Nakamura

TL;DR: The findings that HNE strongly induced intracellular peroxide production, HNE-induced JNK activation was inhibited by pretreatment of the cells with a thiol antioxidant, N-acetylcysteine, and H2O2 significantly activated JNK support the hypothesis that pro-oxidants play a crucial role in the H NE-induced activation of stress signaling pathways.

TL;DR: The aim of this review is to provide an overview of the recent progress on role of ROS derived from NADPH oxidase and redox signaling events involved in angiogenesis, and to provide insight into the NADPH oxidation andRedox signaling components as potential therapeutic targets for tumorAngiogenesis.

TL;DR: B nobiletin is a functionally novel and possible chemopreventive agent in inflammation-associated tumorigenesis and Nobiletin significantly inhibited two distinct stages of skin inflammation induced by double TPA application by decreasing the inflammatory parameters.

TL;DR: The results suggest that MeJA triggers stomatal closing via a receptor distinct from the ABA receptor and that the coi1 mutation disrupts Me JA signaling upstream of the blanch point of ABA signaling and MeJA signaling in Arabidopsis guard cells.

TL;DR: It is suggested that proline offered greater protection against salt stress than betaine did because proline was more effective in increasing the activity of enzymes involved in the antioxidant system.