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Yu Ji
Researcher at University of California, Davis
Publications - 9
Citations - 320
Yu Ji is an academic researcher from University of California, Davis. The author has contributed to research in topics: Wnt signaling pathway & Neurulation. The author has an hindex of 6, co-authored 8 publications receiving 157 citations.
Papers
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Journal ArticleDOI
Impaired neurodevelopmental pathways in autism spectrum disorder: a review of signaling mechanisms and crosstalk.
TL;DR: How various genetic and non-genetic factors which have been reported contributing to ASD interact with WNT, BMP/TGF-β, SHH, FGF, and RA signaling pathways are summarized.
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Wnt signaling in orofacial clefts: crosstalk, pathogenesis and models
Kurt Reynolds,Priyanka Kumari,Lessly Sepulveda Rincon,Ran Gu,Yu Ji,Santosh Kumar,Chengji J. Zhou,Chengji J. Zhou +7 more
TL;DR: Investigation of the crosstalk between the Wnt and other signaling pathways will improve understanding of orofacial cleft development and provide opportunities for treatment and prevention.
Journal ArticleDOI
Genetics and signaling mechanisms of orofacial clefts
TL;DR: Many of the genetic risk factors that may contribute to the presentation of orofacial clefts in patients are discussed and several of the key signaling pathways and underlying cellular mechanisms that control lip and palate formation, as identified primarily through investigating equivalent processes in animal models, are examined.
Journal ArticleDOI
Wnt Signaling in Neural Crest Ontogenesis and Oncogenesis
TL;DR: This review summarizes current understanding of Wnt signaling in NC cell induction, delamination, migration, multipotency, and fate determination, as well as in NC-derived cancers.
Journal ArticleDOI
Cellular and developmental basis of orofacial clefts.
Yu Ji,Michael A. Garland,Bo Sun,Shuwen Zhang,Kurt Reynolds,Moira McMahon,Ratheya Rajakumar,Mohammad S. Islam,Yue Liu,YiPing Chen,Chengji J. Zhou +10 more
TL;DR: The latest cellular and developmental findings may provide a basis for better understanding of the underlying genetic, epigenetic, environmental, and molecular mechanisms of OFCs.