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Yukihiro Yokoyama

Researcher at Nagoya University

Publications -  342
Citations -  10921

Yukihiro Yokoyama is an academic researcher from Nagoya University. The author has contributed to research in topics: Hepatectomy & Bile duct. The author has an hindex of 43, co-authored 319 publications receiving 8835 citations. Previous affiliations of Yukihiro Yokoyama include University of North Carolina at Chapel Hill & University of North Carolina at Charlotte.

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Intraoperative bacterial translocation detected by bacterium-specific ribosomal rna-targeted reverse-transcriptase polymerase chain reaction for the mesenteric lymph node strongly predicts postoperative infectious complications after major hepatectomy for biliary malignancies.

TL;DR: Intraoperative analysis of MLNs (especially MLN-2) by rRNA-targeted RT-qPCR can strongly predict the occurrence of POIC after hepatectomy for biliary malignancy and can select the group of patients with high risk for POIC.
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Adipose tissue-derived mesenchymal stem cell transplantation promotes hepatic regeneration after hepatic ischemia-reperfusion and subsequent hepatectomy in rats

TL;DR: Results indicate that ADSC transplantation may provide beneficial effects in the process of liver regeneration after hepatic ischemia-reperfusion and subsequent hepatectomy.
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Review of hepatopancreatoduodenectomy for biliary cancer: an extended radical approach of Japanese origin.

TL;DR: HPD can be performed with low mortality and offers a better probability of long‐term survival in patients with cholangiocarcinomas that are otherwise unresectable, and should be considered HPD to be a standard approach for laterally advanced cholANGiOCarcinoma.
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Role of thromboxane in producing hepatic injury during hepatic stress.

TL;DR: This review article summarizes the recent knowledge of the role of thromboxane in various types of hepatic stress and the effects of thROMboxane inhibitors in these models.
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Insights into the role of interleukin-6 in the induction of hepatic injury after trauma-hemorrhagic shock

TL;DR: IL-6 plays a significant role in the induction of hepatic dysfunction and liver injury after T-H that appears to be in part mediated by increased thromboxane A(2) levels.