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Journal ArticleDOI

Cocaine-Induced Coronary-Artery Vasoconstriction

Elizabeth E. Gresch
- 01 Apr 1990 - 
- Vol. 32, Iss: 4, pp 286-287
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This article is published in Journal of Occupational and Environmental Medicine.The article was published on 1990-04-01. It has received 12 citations till now. The article focuses on the topics: Vasodilation & Vasoconstriction.

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Citations
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Journal ArticleDOI

Current concepts in resuscitation of patients with local anesthetic cardiac toxicity

TL;DR: Local anesthetics can interfere with cellular metabolism and homeostasis, particularly cell signalling systems and energy transduction pathways, and potentially interact with many different cell membranes, organelles, and a host of membrane-bound and cytosolic, charged molecules.
Journal ArticleDOI

Cardiovascular Mitochondrial Dysfunction Induced by Cocaine: Biomarkers and Possible Beneficial Effects of Modulators of Oxidative Stress.

TL;DR: The comprehension of the mechanisms involving mitochondrial dysfunction could help in selecting the most appropriate mitochondria injury biological marker, such as superoxide dismutase-2 activity and glutathionylated hemoglobin.
Journal ArticleDOI

Cardiovascular and Hepatic Toxicity of Cocaine: Potential Beneficial Effects of Modulators of Oxidative Stress.

TL;DR: It is concluded that available OS modulators do not appear to have clinical efficacy in the treatment of cocaine toxicity.
Journal ArticleDOI

Cocaine-Induced Peripheral Vascular Occlusive Disease: A Case Report

TL;DR: Two patients with cocaine-induced peripheral vascular occlusive disease are presented; one developed progressive bilateral gangrene of both legs necessitating bilateral below-knee amputations, and the second developed similar symptoms after smoking cocaine.
References
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Journal ArticleDOI

Current concepts in resuscitation of patients with local anesthetic cardiac toxicity

TL;DR: Local anesthetics can interfere with cellular metabolism and homeostasis, particularly cell signalling systems and energy transduction pathways, and potentially interact with many different cell membranes, organelles, and a host of membrane-bound and cytosolic, charged molecules.
Journal ArticleDOI

Cardiovascular Mitochondrial Dysfunction Induced by Cocaine: Biomarkers and Possible Beneficial Effects of Modulators of Oxidative Stress.

TL;DR: The comprehension of the mechanisms involving mitochondrial dysfunction could help in selecting the most appropriate mitochondria injury biological marker, such as superoxide dismutase-2 activity and glutathionylated hemoglobin.
Journal ArticleDOI

Cardiovascular and Hepatic Toxicity of Cocaine: Potential Beneficial Effects of Modulators of Oxidative Stress.

TL;DR: It is concluded that available OS modulators do not appear to have clinical efficacy in the treatment of cocaine toxicity.
Journal ArticleDOI

Cocaine-Induced Peripheral Vascular Occlusive Disease: A Case Report

TL;DR: Two patients with cocaine-induced peripheral vascular occlusive disease are presented; one developed progressive bilateral gangrene of both legs necessitating bilateral below-knee amputations, and the second developed similar symptoms after smoking cocaine.
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