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These events can contribute to renal injury with high salt intake in SSR.
The significant decrease in mean arterial pressure in nonsodium‐retaining cirrhotic rats suggests that peripheral vasodilatation is the initial event in the development of salt retention in this model of cirrhosis.
The augmented elimination of salt during ECVE in patients with PBC may explain the rarity of ascites and edema in this variety of cirrhosis.
This observation could be relevant to patients with cirrhosis.