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A. K. M. Shamsuddin

Researcher at University of California, San Diego

Publications -  16
Citations -  403

A. K. M. Shamsuddin is an academic researcher from University of California, San Diego. The author has contributed to research in topics: Cystic fibrosis transmembrane conductance regulator & Optical power. The author has an hindex of 12, co-authored 16 publications receiving 361 citations. Previous affiliations of A. K. M. Shamsuddin include Kobe University & Chittagong Medical College Hospital.

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Journal ArticleDOI

β-adrenergic sweat secretion as a diagnostic test for cystic fibrosis.

TL;DR: β-adrenergic sweat secretion rate determined by evaporimetry is an accurate and reliable technique to assess different levels of CFTR function and to identify patients with CF.
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Enhanced heat loss responses induced by short-term endurance training in exercising women.

TL;DR: It is indicated that physical training improves heat loss responses by decreasing the threshold temperatures and that these effects occur within a month of training and disappearWithin a month after cessation of training.
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Native small airways secrete bicarbonate.

TL;DR: Two separate components for HCO3(-) secretion, likely via CFTR- and calcium-activated chloride channel-dependent processes, are physiologically regulated for likely roles in mucus clearance and antimicrobial innate defenses of small airways.
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Surface fluid absorption and secretion in small airways

TL;DR: A new model proposes that the accordion‐like structure of folds and pleats in the epithelial lining of the airways serves to secrete fluid within the pleats and to absorb fluid along the folds so that fluid levels on the airway surfaces are maintained automatically and airways do not flood or become too dry.
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Changes in blood flow in a conduit artery and superficial vein of the upper arm during passive heating in humans

TL;DR: Blood flow in the brachial artery and the basilic vein increased linearly as the internal temperature variable Tor increased ≤0.6°C, and in both vessels, the passive heating-induced increases in blood flow resulted primarily from achange in blood velocity, rather than from a change in diameter.