A
Alan Leviton
Researcher at Boston Children's Hospital
Publications - 472
Citations - 31540
Alan Leviton is an academic researcher from Boston Children's Hospital. The author has contributed to research in topics: Gestational age & Pregnancy. The author has an hindex of 80, co-authored 468 publications receiving 29272 citations. Previous affiliations of Alan Leviton include Floating Hospital for Children & Tufts Medical Center.
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Journal ArticleDOI
Early blood gas predictors of bronchopulmonary dysplasia in extremely low gestational age newborns.
Sudhir Sriram,Joy Condie,Michael D. Schreiber,Daniel G. Batton,Bhavesh Shah,Carl L. Bose,Matthew M. Laughon,Linda J. Van Marter,Elizabeth N. Allred,Alan Leviton +9 more
TL;DR: Among ELGANs, recurrent/persistent blood gas abnormalities in the first three postnatal days convey information about the risk of severe and very severe BPD.
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Clinical Practice Guidelines and Practice Parameters for the Child Neurologist
TL;DR: This overview, written for child neurologists, addresses such issues as what are clinical practice guidelines, why are they needed, how are they created, how well are they accepted and adhered to, what influences acceptance and adherence.
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Placental cytokine expression in preterm labour and the fetal inflammatory response.
Olaf Dammann,Alan Leviton +1 more
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Antecedents of epilepsy and seizures among children born at extremely low gestational age.
Rachana Singh,Laurie M. Douglass,Thomas M. O'Shea,Carl E. Stafstrom,Elizabeth N. Allred,Steve Engelke,Bhavesh Shah,Alan Leviton,Alan Leviton,Hereen Tc,Kuban Kck +10 more
TL;DR: In children born extremely preterm, epilepsy and seizures not associated with epilepsy have different risk profiles, and though both profiles included indicators of infection and inflammation, the profile of risk factors for epilepsy included multiple indicators of endogenous vulnerability.
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Brain development and the attention spectrum.
TL;DR: Attention Deficit Hyperactivity Disorder (ADHD) seems to be considerably more common than other diagnoses among “double-diagnoses” given to children with developmental dysfunctions, yet the exact prevalence, neurobiological mechanisms, genetic and epigenetic modifications, diagnostic difficulties and treatment methods have not been clearly identified or quantified.