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Allan L. Reiss

Researcher at Stanford University

Publications -  553
Citations -  64704

Allan L. Reiss is an academic researcher from Stanford University. The author has contributed to research in topics: Fragile X syndrome & Autism. The author has an hindex of 118, co-authored 529 publications receiving 59363 citations. Previous affiliations of Allan L. Reiss include Johns Hopkins University School of Medicine & University of California, San Diego.

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Dissociable Intrinsic Connectivity Networks for Salience Processing and Executive Control

TL;DR: Two distinct networks typically coactivated during functional MRI tasks are identified, anchored by dorsal anterior cingulate and orbital frontoinsular cortices with robust connectivity to subcortical and limbic structures, and an “executive-control network” that links dorsolateral frontal and parietal neocortices.
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Functional connectivity in the resting brain: A network analysis of the default mode hypothesis

TL;DR: This study constitutes, to the knowledge, the first resting-state connectivity analysis of the default mode and provides the most compelling evidence to date for the existence of a cohesive default mode network.
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Default-mode network activity distinguishes Alzheimer's disease from healthy aging: Evidence from functional MRI

TL;DR: A goodness-of-fit analysis applied at the individual subject level suggests that activity in the default-mode network may ultimately prove a sensitive and specific biomarker for incipient AD.
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Resting-State Functional Connectivity in Major Depression: Abnormally Increased Contributions from Subgenual Cingulate Cortex and Thalamus

TL;DR: The findings provide cross-modality confirmation of PET studies demonstrating increased thalamic and subgenual cingulate activity in major depression and suggest that a quantitative, resting-state fMRI measure could be used to guide therapy in individual subjects.
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Error-related brain activation during a Go/NoGo response inhibition task.

TL;DR: Investigation of error‐related brain activity associated with failure to inhibit response during a Go/NoGo task provides evidence for a distributed error processing system in the human brain that overlaps partially, but not completely, with brain regions involved in response inhibition and competition.