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Amaal Eman Abdulle

Researcher at University Medical Center Groningen

Publications -  30
Citations -  1029

Amaal Eman Abdulle is an academic researcher from University Medical Center Groningen. The author has contributed to research in topics: Population & Internal medicine. The author has an hindex of 9, co-authored 21 publications receiving 532 citations. Previous affiliations of Amaal Eman Abdulle include University of Groningen.

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Angiotensin?converting enzyme 2 (ACE2), SARS?CoV?2 and the pathophysiology of coronavirus disease 2019 (COVID?19)

TL;DR: The role of ACE2 in CO VID‐19 pathophysiology is described, including factors influencing ACE2 expression and activity in relation to COVID‐19 severity, and the relevant pathological changes resulting from SARS‐CoV‐2 infection are discussed.
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COVID-19: immunopathology, pathophysiological mechanisms, and treatment options.

TL;DR: A review of the main pathological changes observed in COVID-19 within the most commonly affected organ systems, with special emphasis on immunopathology, is provided in this paper, which provides valuable information with regard to our understanding of pathophysiology as well as the development of evidence-based treatment regimens.
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Early Predictors for Long-Term Functional Outcome After Mild Traumatic Brain Injury in Frail Elderly Patients.

TL;DR: The frail elderly had worse long-term outcome, and early complaints were found to be a stronger predictor of unfavorable outcome than age, whereas age was not a significant predictor.
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The Role of Oxidative Stress in the Development of Systemic Sclerosis Related Vasculopathy

TL;DR: The background of oxidative stress related processes (e.g., EC injury, autoimmunity, inflammation, and vascular wall remodeling) that may contribute to SSc vasculopathy are presented and a potential role of ROS in the initiation and progression of SSc Vasculopathy is suggested.
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N-Acetylcysteine and Hydrogen Sulfide in Coronavirus Disease 2019.

TL;DR: In this article, the role of hydrogen sulfide (H2S) in the treatment of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) was investigated.