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Andreas Meyer-Lindenberg

Researcher at Heidelberg University

Publications -  642
Citations -  51374

Andreas Meyer-Lindenberg is an academic researcher from Heidelberg University. The author has contributed to research in topics: Medicine & Schizophrenia. The author has an hindex of 107, co-authored 534 publications receiving 44592 citations. Previous affiliations of Andreas Meyer-Lindenberg include United States Department of Health and Human Services & University of Mannheim.

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5-HTTLPR polymorphism impacts human cingulate-amygdala interactions: a genetic susceptibility mechanism for depression

TL;DR: Morphometrical analyses showed reduced gray matter volume in short-allele carriers in limbic regions critical for processing of negative emotion, particularly perigenual cingulate and amygdala, and relative uncoupling of this circuit.
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Oxytocin Modulates Neural Circuitry for Social Cognition and Fear in Humans

TL;DR: It is shown that human amygdala function is strongly modulated by oxytocin, and this results indicate a neural mechanism for the effects of Oxytocin in social cognition in the human brain and provide a methodology and rationale for exploring therapeutic strategies in disorders in which abnormal amygdala function has been implicated, such as social phobia or autism.
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Oxytocin and vasopressin in the human brain: social neuropeptides for translational medicine

TL;DR: OXT and AVP are emerging as targets for novel treatment approaches — particularly in synergistic combination with psychotherapy — for mental disorders characterized by social dysfunction, such as autism, social anxiety disorder, borderline personality disorder and schizophrenia.
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City living and urban upbringing affect neural social stress processing in humans.

TL;DR: It is shown that urban upbringing and city living have dissociable impacts on social evaluative stress processing in humans, and distinct neural mechanisms for an established environmental risk factor are identified.
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Hierarchical Organization of Human Cortical Networks in Health and Schizophrenia

TL;DR: It is proposed that the topological differences between divisions of normal cortex may represent the outcome of different growth processes for multimodal and transmodal networks and that neurodevelopmental abnormalities in schizophrenia specifically impact multi-modal cortical organization.