Peter Kirsch

TL;DR: It is shown that human amygdala function is strongly modulated by oxytocin, and this results indicate a neural mechanism for the effects of Oxytocin in social cognition in the human brain and provide a methodology and rationale for exploring therapeutic strategies in disorders in which abnormal amygdala function has been implicated, such as social phobia or autism.

TL;DR: OXT and AVP are emerging as targets for novel treatment approaches — particularly in synergistic combination with psychotherapy — for mental disorders characterized by social dysfunction, such as autism, social anxiety disorder, borderline personality disorder and schizophrenia.

TL;DR: It is shown that urban upbringing and city living have dissociable impacts on social evaluative stress processing in humans, and distinct neural mechanisms for an established environmental risk factor are identified.

TL;DR: A meta-analysis of all available neuroimaging studies of rs4680 shows significant association between the COMT genotype and prefrontal activation, and meta-analytical evidence for a neural substrate for the pleiotropic behavioral effects of COMT genetic variation is found.