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Andrew C.C. Wong
Researcher at University of Chicago
Publications - 4
Citations - 1125
Andrew C.C. Wong is an academic researcher from University of Chicago. The author has contributed to research in topics: Zebrafish & Cellular differentiation. The author has an hindex of 4, co-authored 4 publications receiving 1062 citations. Previous affiliations of Andrew C.C. Wong include Pennsylvania State University & Emory University.
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Journal ArticleDOI
SLC24A5, a Putative Cation Exchanger, Affects Pigmentation in Zebrafish and Humans
Rebecca L. Lamason,Manzoor Ali P.K. Mohideen,Manzoor Ali P.K. Mohideen,Jason R. Mest,Andrew C.C. Wong,Andrew C.C. Wong,Heather L. Norton,Michele C. Aros,Michael J. Jurynec,Xianyun Mao,Vanessa R. Humphreville,Jasper E. Humbert,Jasper E. Humbert,Soniya Sinha,Jessica L. Moore,Jessica L. Moore,Pudur Jagadeeswaran,Wei Zhao,Gang Ning,Izabela Makalowska,Paul M. McKeigue,David H. O'Donnell,Rick A. Kittles,Esteban J. Parra,N. J. Mangini,David Grunwald,Mark D. Shriver,Victor A. Canfield,Keith C. Cheng +28 more
TL;DR: It is shown that zebrafish golden mutants share these melanosomal changes and that golden encodes a putative cation exchanger slc24a5 (nckx5) that localizes to an intracellular membrane, likely the melanosome or its precursor.
Journal ArticleDOI
“Molecular rulers” for calibrating phenotypic effects of telomere imbalance
Christa Lese Martin,Darrel Waggoner,Andrew C.C. Wong,S Uhrig,Jessica A. Roseberry,J Hedrick,Svetlana Pack,Karen L. Russell,Elaine H. Zackai,William B. Dobyns,David H. Ledbetter +10 more
TL;DR: Developing “molecular rulers” that incorporate multiple clones at each telomere that span the most distal 5 Mb region will allow better phenotype prediction and prognosis related to telomeric imbalances identified in normal people.
Journal ArticleDOI
The evolutionary origin of human subtelomeric homologies--or where the ends begin.
TL;DR: The evolutionary origin of seven subtelomeric clones is studied by performing comparative FISH analysis on a primate panel that included great apes and Old World monkeys, demonstrating that these segmental duplications are most likely the result of ancestral unbalanced translocations that have been fixed in the genome during recent primate evolution.
Journal ArticleDOI
Histology-based screen for zebrafish mutants with abnormal cell differentiation.
Manzoor Ali P.K. Mohideen,Lee G. Beckwith,Gladys S. Tsao-Wu,Jessica L. Moore,Andrew C.C. Wong,Mala R. Chinoy,Keith C. Cheng +6 more
TL;DR: This mutagenesis screen in zebrafish is proof of principle for the feasibility of histologic screens to yield novel mutations, including potential models of human disease.