Andrew P. Dobson

TL;DR: Whether host transfer has been a repeated event between the original poultry hosts and house finches, whether only a single host transfer was ultimately responsible for the emergence of M. gallisepticum, and whether the spread of the pathogen from east to west across North America has resulted in spatial structuring in the pathogenic.

TL;DR: The paper and its commentary emphasize that this model provides a basis for the transmission pathway known as “human-to-human” and demonstrates its importance, relative to the “environment- to- human” pathway, in the ‘explosive’ character of cholera epidemics.

TL;DR: Comparisons of worm burdens from single and mixed species infections within individual hosts suggest that interactions between parasite species only rarely leads to reduced worm burdens.

TL;DR: The results indicate that spatial heterogeneities in virus clearance, virus pathogenicity or both, resulting from the unique structure of the respiratory tract, may drive optimal receptor binding affinity–that maximizes influenza virus reproductive fitness at the population level–towards sialic acids with α2,6 linkage to galactose.

TL;DR: The results show that highly aggregated parasite species are more likely to coexist, but are less able to regulate their host population.