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Andrey Mazarati

Researcher at University of California, Los Angeles

Publications -  112
Citations -  6080

Andrey Mazarati is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Epilepsy & Status epilepticus. The author has an hindex of 43, co-authored 107 publications receiving 5586 citations. Previous affiliations of Andrey Mazarati include Boston Children's Hospital & Veterans Health Administration.

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Epilepsy and brain inflammation.

TL;DR: The clinical observations in drug-resistant human epilepsies and the experimental findings in adult and immature rodents linking brain inflammation to the epileptic process in a causal and reciprocal manner are reported.
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Patterns of Status Epilepticus-Induced Neuronal Injury during Development and Long-Term Consequences

TL;DR: The results provide strong evidence for the vulnerability of the immature brain to seizure-induced damage, which bears features of both necrotic and apoptotic death and contributes to synaptic reorganization and the development of chronic epilepsy.
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Time dependent decrease in the effectiveness of antiepileptic drugs during the course of self-sustaining status epilepticus

TL;DR: It is concluded that antiepileptic drugs, while highly effective in blocking the induction of SSSE, failed to affect its maintenance.
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Modulation of Hippocampal Excitability and Seizures by Galanin

TL;DR: The data provide further evidence that hippocampal Galanin acts as an endogenous anticonvulsant and suggest that genetically induced changes in galanin expression modulate both hippocampal excitability and predisposition to epileptic seizures.
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Depression after status epilepticus: behavioural and biochemical deficits and effects of fluoxetine.

TL;DR: Initial evidence is provided that post-SE model of TLE might serve as a model of the comorbidity of epilepsy and depression, and the finding that behavioural equivalents of depression were resistant to an antidepressant medication suggested that depression in epilepsy might have distinct underlying mechanisms beyond alterations in serotonergic pathways.