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Angelo Rosario Carotenuto

Researcher at University of Naples Federico II

Publications -  31
Citations -  407

Angelo Rosario Carotenuto is an academic researcher from University of Naples Federico II. The author has contributed to research in topics: Mechanobiology & Medicine. The author has an hindex of 9, co-authored 24 publications receiving 255 citations.

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Cells competition in tumor growth poroelasticity

TL;DR: In this paper, an enhanced multi-scale poroelastic model undergoing large deformations and embodying inelastic growth is proposed, where the net growth terms directly result from the "interspecific" predator-prey competition occurring at the micro-scale level between healthy and abnormal cell species.
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Stress-shielding, growth and remodeling of pulmonary artery reinforced with copolymer scaffold and transposed into aortic position

TL;DR: A mathematical model is developed in order to simulate the biomechanical changes in pulmonary artery subjected to systemic pressure load and reinforced with a combination of resorbable and auxetic synthetic materials, and theoretical outcomes show that a virtuous biomechanicals cooperation between biological and synthetic materials takes place, determining the overall success of the autograft system.
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Compliance mismatch and compressive wall stresses drive anomalous remodelling of pulmonary trunks reinforced with Dacron grafts

TL;DR: In vivo observations of the physical interaction among aorta, autograft and widely adopted synthetic (Dacron) prostheses utilized in transposition of pulmonary artery demonstrate the effectiveness of the proposed biomechanical hypothesis and open the way for possible engineering-guided strategies to support and optimize surgical procedures.
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Buckling soft tensegrities: Fickle elasticity and configurational switching in living cells

TL;DR: In this article, a soft-strut tense-grity model is proposed, which takes into account geometrical nonlinearity (i.e. large deformations) and hyper-elasticity of both tendons and bars.
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Biomechanics drive histological wall remodeling of neoaortic root: A mathematical model to study the expression levels of ki 67, metalloprotease, and apoptosis transition.

TL;DR: An histological remodeling phenomenon sustained by increased level of MMP-9, augmented cell proliferation and reduced apoptosis in the reinforced PA was demonstrated and the mathematical model predicted the biomechanical behavior subtended by the histological changes of the PA in these settings.