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Anthony V. Furano
Researcher at National Institutes of Health
Publications - 66
Citations - 3284
Anthony V. Furano is an academic researcher from National Institutes of Health. The author has contributed to research in topics: DNA & Retrotransposon. The author has an hindex of 33, co-authored 63 publications receiving 3086 citations. Previous affiliations of Anthony V. Furano include Georgetown University Medical Center.
Papers
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Journal ArticleDOI
L1 (LINE-1) Retrotransposon Evolution and Amplification in Recent Human History
TL;DR: The successive emergence and amplification of distinct Ta L1 subfamilies shows that L1 evolution has been as active in recent human history as it has been found to be for rodent L1 families.
Journal ArticleDOI
Selection Against Deleterious LINE-1-Containing Loci in the Human Lineage
TL;DR: It is concluded that most FL L1s were deleterious and thus subject to purifying selection and that genetic damage produced by both L1 retrotransposition and ectopic recombination between L1 elements could provide the basis for their negative selection.
Book ChapterDOI
The biological properties and evolutionary dynamics of mammalian LINE-1 retrotransposons.
TL;DR: The structure and biological properties of L1 elements are reviewed, including their regulation, replication, evolution, and interaction with their mammalian hosts, indicating that they represent challenging and fascinating biological phenomena.
Journal ArticleDOI
Content of elongation factor Tu in Escherichia coli
TL;DR: The content of elongation factor Tu in E. coli B has been determined both by radioimmune assay and by GDP binding, and levels resemble those reported for tRNA, in contrast with the 1:1 ratio of factor to ribosomes reported for elongation factors Ts and G.
Journal ArticleDOI
NGF stimulates incorporation of fucose or glucosamine into an external glycoprotein in cultured rat PC12 pheochromocytoma cells
TL;DR: Despite their similarities in apparent size and exposure on cell surfaces, the NILE and LETS glycoproteins are shown to be immunologically distinct.