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Antje Bock

Researcher at Charité

Publications -  7
Citations -  315

Antje Bock is an academic researcher from Charité. The author has contributed to research in topics: Deep brain stimulation & Subthalamic nucleus. The author has an hindex of 6, co-authored 7 publications receiving 227 citations.

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Journal ArticleDOI

Cortico-pallidal oscillatory connectivity in patients with dystonia

TL;DR: Pallido-cerebellar oscillatory connectivity and its association with dystonic symptoms provides further confirmation of cerebellar involvement in dystonia that has been recently reported using functional magnetic resonance imaging and fibre tracking.
Journal ArticleDOI

Dopamine-dependent scaling of subthalamic gamma bursts with movement velocity in patients with Parkinson's disease.

TL;DR: It is proposed that insufficient recruitment of fast gamma bursts during movement may underlie bradykinesia as one of the cardinal symptoms in Parkinson’s disease.
Journal ArticleDOI

Thalamic gamma oscillations correlate with reaction time in a Go/noGo task in patients with essential tremor.

TL;DR: The role of gamma activity as a physiological prokinetic activity in the motor system is supported and subtle fluctuations in pre-cue gamma band activity may have an impact on task performance and may index arousal-related states.
Proceedings ArticleDOI

Coherence and imaginary part of coherency identifies cortico-muscular and cortico-thalamic coupling

TL;DR: The COH and iCOH between magnetoencephalographic (MEG) and electromyographic (EMG) signals of the healthy subjects yields the expected result for cortico-muscular coupling and suggests that volume conduction is not important for the analysis of interactions between MEG and bipolar STN electrodes.
Journal ArticleDOI

Subthalamic and pallidal oscillatory activity in patients with Neurodegeneration with Brain Iron Accumulation type I (NBIA-I)

TL;DR: The oscillatory activity pattern in the STN and the GPi is shown for the first time in juvenile patients with dystonia plus syndrome due to NBIA-I to support the link between hyperkinetic motor symptoms such as dystonIA and enhanced basal ganglia low frequency activity irrespective of the underlying etiology of dySTONia.