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April D. Strader
Researcher at Southern Illinois University Carbondale
Publications - 21
Citations - 1573
April D. Strader is an academic researcher from Southern Illinois University Carbondale. The author has contributed to research in topics: Leptin & White adipose tissue. The author has an hindex of 14, co-authored 21 publications receiving 1462 citations. Previous affiliations of April D. Strader include Southern Illinois University School of Medicine & University of Cincinnati.
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Journal ArticleDOI
Gastrointestinal hormones and food intake.
TL;DR: Although traditionally thought of as short-term, meal-related signals, enhanced, chronic hormone secretion and signaling resulting from gut reconstruction as seen with gastric bypass surgery most likely contributes to the superior efficacy of surgery as a treatment for obesity.
Journal ArticleDOI
Weight loss through ileal transposition is accompanied by increased ileal hormone secretion and synthesis in rats
April D. Strader,Torsten P. Vahl,Ronald J. Jandacek,Stephen C. Woods,David A. D'Alessio,Randy J. Seeley +5 more
TL;DR: The association of weight loss with increased release of GLP-1 and PYY suggests that procedures that promote gastrointestinal endocrine function can reduce energy intake and support the importance of evaluating the contribution of gastrointestinal hormones to the weight loss seen with bariatric surgery.
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Estradiol-Dependent Decrease in the Orexigenic Potency of Ghrelin in Female Rats
Deborah J. Clegg,Lynda M. Brown,Jeffrey M. Zigman,Christopher J. Kemp,April D. Strader,Stephen C. Benoit,Stephen C. Woods,Michela Mangiaracina,Nori Geary,Nori Geary +9 more
TL;DR: It is suggested that estradiol inhibits the orexigenic action of ghrelin in females, that weight gain associated with OVX is gh Relin mediated, and that this endocrine interaction may account for an important sex differences in food intake and the regulation of body weight.
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Mice lacking the syndecan-3 gene are resistant to diet-induced obesity
TL;DR: The data implicate syndecan-3 in the regulation of body weight and suggest that inhibition of syndecans may provide a therapeutic approach for the treatment of obesity resulting from exposure to high-fat diets.
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The effect of fat removal on glucose tolerance is depot specific in male and female mice
TL;DR: It is concluded that WAT contribution to glucose homeostasis is depot specific, with male gonadal EWAT contributing to glucoseHomeostasis in the lean state, whereas female gonadal PWAT contributes to glucosehomeostasisIn both lean and obese mice.