S
Stephen C. Woods
Researcher at University of Cincinnati
Publications - 584
Citations - 53408
Stephen C. Woods is an academic researcher from University of Cincinnati. The author has contributed to research in topics: Insulin & Medicine. The author has an hindex of 118, co-authored 517 publications receiving 50306 citations. Previous affiliations of Stephen C. Woods include University of Washington & Johns Hopkins University School of Medicine.
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Journal ArticleDOI
Central nervous system control of food intake
TL;DR: A model is described that delineates the roles of individual hormonal and neuropeptide signalling pathways in the control of food intake and the means by which obesity can arise from inherited or acquired defects in their function.
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Signals That Regulate Food Intake and Energy Homeostasis
TL;DR: This review focuses on the molecular signals that modulate food intake while integrating the body's immediate and long-term energy needs.
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Hypothalamic mTOR Signaling Regulates Food Intake
Daniela Cota,Karine Proulx,Kathi Smith,Sara C. Kozma,George Thomas,Stephen C. Woods,Randy J. Seeley +6 more
TL;DR: It is demonstrated that mTOR signaling plays a role in the brain mechanisms that respond to nutrient availability, regulating energy balance, and that hypothalamic activity is directly tied to the regulation of energy intake.
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Chronic intracerebroventricular infusion of insulin reduces food intake and body weight of baboons
Stephen C. Woods,Stephen C. Woods,Elizabeth C. Lotter,Elizabeth C. Lotter,L. David McKay,L. David McKay,Daniel Porte,Daniel Porte +7 more
TL;DR: Additional evidence is presented by showing that in baboons the infusion of exogenous insulin into the CSF elicits a reliable and predictable decrease in food intake and body weight.
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Specificity of Leptin Action on Elevated Blood Glucose Levels and Hypothalamic Neuropeptide Y Gene Expression in ob/ob Mice
Michael W. Schwartz,Denis G. Baskin,Thomas R. Bukowski,Joseph L. Kuijper,Donald C. Foster,Gerry Lasser,Donna E. Prunkard,Daniel Porte,Stephen C. Woods,Randy J. Seeley,David S. Weigle +10 more
TL;DR: In ob/ob mice, systemic administration of leptin inhibits NPY gene overexpression through a specific action in the arcuate nucleus and exerts a hypoglycemic action that is partly independent of its weight-reducing effects.