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Arthur A. Vandenbark
Researcher at Oregon Health & Science University
Publications - 134
Citations - 7372
Arthur A. Vandenbark is an academic researcher from Oregon Health & Science University. The author has contributed to research in topics: Experimental autoimmune encephalomyelitis & T cell. The author has an hindex of 41, co-authored 134 publications receiving 6920 citations. Previous affiliations of Arthur A. Vandenbark include Veterans Health Administration & United States Department of Veterans Affairs.
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Journal ArticleDOI
Therapy with Recombinant T-cell Receptor Ligand reduces infarct size and infiltrating inflammatory cells in brain after middle cerebral artery occlusion in mice
Suzan Dziennis,Sarah L. Mader,Kozaburo Akiyoshi,Xuefang Ren,Patricia Ayala,Gregory G. Burrows,Arthur A. Vandenbark,Arthur A. Vandenbark,Paco S. Herson,Patricia D. Hurn,Halina Offner +10 more
TL;DR: Effective treatment of experimental stroke with RTL551 within a therapeutically relevant 4 h time window through immune regulation of myelin-reactive inflammatory T-cells is demonstrated.
Journal ArticleDOI
Ethinyl estradiol treats collagen-induced arthritis in DBA/1LacJ mice by inhibiting the production of TNF-α and IL-1β
Sandhya Subramanian,Micah Tovey,Michael Afentoulis,Aric Krogstad,Arthur A. Vandenbark,Arthur A. Vandenbark,Halina Offner,Halina Offner +7 more
TL;DR: The effectiveness of ethinyl estradiol in treating collagen-induced arthritis (CIA) induced with bovine type II collagen (bCII) in DBA/1LacJ mice, a CIA susceptible strain is reported, supporting its possible clinical use for treating rheumatoid arthritis in humans.
Journal ArticleDOI
Effects of cytokine deficiency on chemokine expression in CNS of mice with EAE.
Agata Matejuk,Agata Matejuk,Agata Matejuk,Jami Dwyer,Atsushi Ito,Atsushi Ito,Zachary Bruender,Arthur A. Vandenbark,Arthur A. Vandenbark,Halina Offner,Halina Offner,Halina Offner +11 more
TL;DR: The results demonstrate that expression of most chemokines/receptors was drastically inhibited in TNF‐α deficient mice, and was reflective of delayed onset and reduced severity of EAE, and implicate MIP‐1α, Mip‐2, and MCP‐1 in controlling CNS inflammation and severity ofEAE.
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Prevention and treatment of relapsing autoimmune encephalomyelitis with myelin peptide-coupled splenocytes
TL;DR: The utility of using neuroantigen‐coupled accessory cells to prevent and treat relapsing EAE is demonstrated, and the powerful protective effect of treating mice before induction of EAE with PLP139–151‐conjugated splenocytes (SPL) is demonstrated.
Journal Article
Human Transfer Factor: Fractionation and Biologic Activity
TL;DR: The data suggest that TF preparations contain components which can affect immune reactions in both specific and nonspecific ways.