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Arthur H. Moser
Researcher at University of California, San Francisco
Publications - 94
Citations - 9057
Arthur H. Moser is an academic researcher from University of California, San Francisco. The author has contributed to research in topics: Tumor necrosis factor alpha & Cholesterol. The author has an hindex of 52, co-authored 94 publications receiving 8554 citations. Previous affiliations of Arthur H. Moser include San Francisco VA Medical Center & United States Department of Veterans Affairs.
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Journal ArticleDOI
Effects of infection and inflammation on lipid and lipoprotein metabolism: mechanisms and consequences to the host.
Weerapan Khovidhunkit,Min Sun Kim,Riaz A. Memon,Judy K. Shigenaga,Arthur H. Moser,Kenneth R. Feingold,Carl Grunfeld +6 more
TL;DR: APR-induced alterations initially protect the host from the harmful effects of bacteria, viruses, and parasites, however, if prolonged, these changes in the structure and function of lipoproteins will contribute to atherogenesis.
Journal ArticleDOI
Interleukin-6 stimulates hepatic triglyceride secretion in rats.
Katsunori Nonogaki,Gerald M. Fuller,Nelson L. Fuentes,Arthur H. Moser,Ilona Staprans,Carl Grunfeld,Kenneth R. Feingold +6 more
TL;DR: It is demonstrated that changes in hepatic triglyceride metabolism are another acute phase response that can be induced by IL-6, and the hypertriglyceridemia induced byIL-6 was due to increased secretion by the liver.
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Leptin deficiency enhances sensitivity to endotoxin-induced lethality
Raffaella Faggioni,Giamila Fantuzzi,Cem Gabay,Arthur H. Moser,Charles A. Dinarello,Kenneth R. Feingold,Carl Grunfeld +6 more
TL;DR: Leptin participates in the host response to inflammation by modulating the host immune and cytokine responses after LPS, and treatment with leptin reversed the increased sensitivity to LPS-induced lethality found in ob/ obmice.
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Up-regulation of peroxisome proliferator-activated receptors (PPAR-alpha) and PPAR-gamma messenger ribonucleic acid expression in the liver in murine obesity: troglitazone induces expression of PPAR-gamma-responsive adipose tissue-specific genes in the liver of obese diabetic mice.
Riaz A. Memon,Laurence H. Tecott,Katsunori Nonogaki,Anne P. Beigneux,Arthur H. Moser,Carl Grunfeld,Kenneth R. Feingold +6 more
TL;DR: The results suggest that the effects of PPAR-gamma activators on lipid metabolism and energy homeostasis in obesity and type 2 diabetes may be partly mediated through their effects on PPAR -gamma in the liver.
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Paraoxonase activity in the serum and hepatic mRNA levels decrease during the acute phase response
TL;DR: It is demonstrated that serum PON activity decreases following endotoxin (LPS) administration in Syrian hamsters, suggesting that the decrease in PON could be another factor linking the acute phase response with increased atherogenesis.