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Artur H. Swiergiel
Researcher at University of Gdańsk
Publications - 71
Citations - 3685
Artur H. Swiergiel is an academic researcher from University of Gdańsk. The author has contributed to research in topics: Chronic stress & Receptor. The author has an hindex of 31, co-authored 69 publications receiving 3426 citations. Previous affiliations of Artur H. Swiergiel include Louisiana State University & LSU Health Sciences Center Shreveport.
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Cytokines as mediators of depression: What can we learn from animal studies?
TL;DR: The value of sickness behavior as an animal model of major depressive disorder is limited, so that care should be taken in extrapolating results from the model to the human disorder, but it is concluded that immune activation and cytokines may be involved in depressive symptoms in some patients.
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Stress-induced behaviors require the corticotropin-releasing hormone (CRH) receptor, but not CRH
Stacie C. Weninger,Adrian J. Dunn,Louis J. Muglia,Pieter Dikkes,Klaus A. Miczek,Artur H. Swiergiel,Craig W. Berridge,Joseph A. Majzoub +7 more
TL;DR: An unidentified CRH-like molecule other than CRH or urocortin, acting through the CRH receptors in brain regions believed to mediate stress-induced behaviors, may mediate the behavioral response to stress, either alone or in concert with CRH.
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Properties of gap junction blockers and their behavioural, cognitive and electrophysiological effects: animal and human studies.
TL;DR: It is concluded that despite a number of different problems, the currently used gap junction blockers could be useful tools in pharmacology and neuroscience.
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Effects of Interleukin-1β and Lipopolysaccharide on Behavior of Mice in the Elevated Plus-Maze and Open Field Tests
TL;DR: Results do not provide unequivocal support for the suggestion that LPS or IL-1 mediate anxiety, but because infections, endotoxins, and the ensuing cytokines cause alterations in CNS norepinephrine and serotonin, they may contribute to emotionality, and perhaps to anxiety.
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Epigenetics of stress adaptations in the brain.
TL;DR: It is concluded that specific epigenetic mechanisms in the CNS are involved in the stress response, including genome-wide epigenetic changes of DNA methylation and particular genes involved in epigenetic responses that participate in the brain response to chronic psychogenic stressors.