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Barbara E. Murray
Researcher at University of Texas at Austin
Publications - 235
Citations - 23158
Barbara E. Murray is an academic researcher from University of Texas at Austin. The author has contributed to research in topics: Enterococcus faecalis & Enterococcus faecium. The author has an hindex of 73, co-authored 232 publications receiving 21704 citations. Previous affiliations of Barbara E. Murray include Emerging Pathogens Institute & University of Texas Health Science Center at Houston.
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In vitro activity of 19 antimicrobial agents against enterococci from healthy subjects and hospitalized patients and use of an ace gene probe from Enterococcus faecalis for species identification.
TL;DR: It is demonstrated that an intragenic probe of the gene ace from E. faecalis showed specific hybridizations to all E. Faecalis isolates, suggesting the usefulness of this gene for identification of this species.
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Rapid Identification of Enterococcus hirae and Enterococcus durans by PCR and Detection of a Homologue of the E. hirae mur-2 Gene in E. durans
Cesar A. Arias,Beatriz Robredo,Kavindra V. Singh,Carmen Torres,Diana Panesso,Barbara E. Murray +5 more
TL;DR: PCR using primers for two genes (copY and murG) of E. hirae strains showed amplification with E. durans strains only, and PCR (under high-stringency conditions) withPrimers for the mur-2ed gene gave the expected amplification product only with E.'s durans strain.
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Enterococcus faecalis rnjB Is Required for Pilin Gene Expression and Biofilm Formation
Peng Gao,Kenneth L. Pinkston,Sreedhar R. Nallapareddy,Ambro van Hoof,Barbara E. Murray,Barrett R. Harvey,Barrett R. Harvey +6 more
TL;DR: The involvement of rnjB in E. faecalis pilin gene expression is demonstrated and insight into a novel mechanism of regulation of pilus production in Gram-positive pathogens is provided.
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Characterization of dihydrofolate reductase genes from trimethoprim-susceptible and trimethoprim-resistant strains of Enterococcus faecalis
TL;DR: Results indicate thatdfrF is an acquired but probably chromosomally located gene which is responsible for in vitro HLR to trimethoprim in E. faecalis, and this gene is presumed to be the intrinsic E. Faecalis dfr gene (which causes resistance in E coli when cloned in multiple copies).
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Evaluation of the molecular epidemiology of an outbreak of multiply resistant Shigella sonnei in a day-care center by using pulsed-field gel electrophoresis and plasmid DNA analysis.
TL;DR: DCC outbreak isolates could be distinguished from most sporadic isolates by antimicrobial susceptibility testing, but plasmid analysis and PFGE could not differentiate common-source isolates from sporadic isolate in the same location during the same time period, indicating that isolates present in the community were genetically similar to those producing outbreaks in the DCC.