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Barbara E. Murray
Researcher at University of Texas at Austin
Publications - 235
Citations - 23158
Barbara E. Murray is an academic researcher from University of Texas at Austin. The author has contributed to research in topics: Enterococcus faecalis & Enterococcus faecium. The author has an hindex of 73, co-authored 232 publications receiving 21704 citations. Previous affiliations of Barbara E. Murray include Emerging Pathogens Institute & University of Texas Health Science Center at Houston.
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Repetitive Sequence-Based PCR versus Pulsed-Field Gel Electrophoresis for Typing of Enterococcus faecalis at the Subspecies Level
TL;DR: Repetitive sequence-based PCR was compared to pulsed-field gel electrophoresis (PFGE) for the ability to discriminateEnterococcus faecalis isolates at the subspecies level and results were more difficult to interpret than PFGE results.
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Evidence that the enterococcal polysaccharide antigen gene (epa) cluster is widespread in Enterococcus faecalis and influences resistance to phagocytic killing of E. faecalis.
TL;DR: The results indicate that the epa gene cluster is widespread among E. faecalis and confers some protection against human host defenses.
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Mechanism for Sortase Localization and the Role of Sortase Localization in Efficient Pilus Assembly in Enterococcus faecalis
Kimberly A. Kline,Andrew L. Kau,Swaine L. Chen,Adeline Lim,Jerome S. Pinkner,Jason W. Rosch,Sreedhar R. Nallapareddy,Barbara E. Murray,Birgitta Henriques-Normark,Wandy L. Beatty,Michael G. Caparon,Scott J. Hultgren +11 more
TL;DR: It is found that a positively charged domain can act as a localization retention signal for the focal compartmentalization of membrane proteins in E. faecalis and abolished both its retention at single foci and its function in efficient pilus assembly.
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Relative Contributions of Ebp Pili and the Collagen Adhesin Ace to Host Extracellular Matrix Protein Adherence and Experimental Urinary Tract Infection by Enterococcus faecalis OG1RF
TL;DR: Data implicate the Ebp pili as having some role in collagen adherence, albeit less than that of Ace, and a very major role in fibrinogen adherence, which may explain in part the importance of these pili in experimental endocarditis.
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Relapse of Type A β-Lactamase-Producing Staphylococcus aureus Native Valve Endocarditis during Cefazolin Therapy: Revisiting the Issue
TL;DR: Analysis of the infecting strain disclosed a high minimum inhibitory concentration of cefazolin when a large inoculum was used, as well as rapid and complete cefzolin degradation, which was associated with regrowth in a time-kill experiment.