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Barbara E. Murray
Researcher at University of Texas at Austin
Publications - 235
Citations - 23158
Barbara E. Murray is an academic researcher from University of Texas at Austin. The author has contributed to research in topics: Enterococcus faecalis & Enterococcus faecium. The author has an hindex of 73, co-authored 232 publications receiving 21704 citations. Previous affiliations of Barbara E. Murray include Emerging Pathogens Institute & University of Texas Health Science Center at Houston.
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Comparative in vitro activities of amoxicillin-clavulanic acid, cefuroxime, cephalexin, and cephalothin against trimethoprim-resistant Escherichia coli isolated from stools of children attending day-care centers.
TL;DR: Although all three oral beta-lactams tested were generally active at concentrations likely to be achieved in urine, cefuroxime and cephalexin were more potent and are thus more likely to been inhibitory at the concentrations needed for systemic infections.
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Chaining in enterococci revisited: correlation between chain length and gelatinase phenotype, and gelE and fsrB genes among clinical isolates of Enterococcus faecalis.
TL;DR: A marked increase in chaining seen in an Enterococcus faecalis OG1RF mutant (TX5128) has been explained by the lack of the GelE protease (gelatinase) suggesting the possibility that chaining among clinical isolates (one-third of which are gelatinase non-producers) might be associated with the absence of GelE.
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Erratum: Influence of origin of isolates, especially endocarditis isolates, and various genes on biofilm formation by Enterococcus faecalis (Infection and Immunity (2004) 72, 6 (3658-3663))
TL;DR: Influence of Origin of Isolates, Especially Endocarditis Isolate, and Various Genes on Biofilm Formation by Enterococcus faecalis is studied.
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Lack of homology of enterococci which have high-level resistance to trimethoprim with the dfrA gene of Staphylococcus aureus.
TL;DR: Acquisition of resistance to TMP is another example of the multiple antimicrobial resistance typically displayed by enterococci.
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Could a Phosphotransferase System Provide the Means to Control Outbreaks of Enterococcus faecium Infection
TL;DR: The epidemiology of vancomycin-resistant E. faecium (VREfm) presented a quandary: at the time, VREfm was alreadyacommon cause of US healthcare-associated infections, such organisms were not found in the community, but concurrently, V REfm was rare as a cause of infection in the European Union.