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Barbara E. Power
Researcher at Commonwealth Scientific and Industrial Research Organisation
Publications - 35
Citations - 2754
Barbara E. Power is an academic researcher from Commonwealth Scientific and Industrial Research Organisation. The author has contributed to research in topics: Antibody & Ribosome display. The author has an hindex of 21, co-authored 35 publications receiving 2661 citations. Previous affiliations of Barbara E. Power include Monash University, Clayton campus.
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DNA hypomethylation and human diseases.
TL;DR: How global demethylation of repeat sequences including transposable elements and the site-specific hypomethylation of certain genes might contribute to the deleterious effects that ultimately result in the initiation and progression of cancer and other diseases is considered.
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Genomic structure of tbe large RNA segment of infectious bursal disease virus
TL;DR: It is proposed that birnaviruses, in particular IBDV, possess monocistronic segments and that the precursor is proteolytically processed in vivo.
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A new generation of protein display scaffolds for molecular recognition
TL;DR: Here, it is predicted the important role of these novel binding reagents as a toolkit for biotechnological and biomedical applications is predicted.
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Dimeric and trimeric antibodies: high avidity scFvs for cancer targeting.
TL;DR: A number of cancer-targeting scFv diabodies that have undergone successful pre-clinical trials for in vivo stability and efficacy are highlighted.
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Adoptive transfer of T cells modified with a humanized chimeric receptor gene inhibits growth of Lewis-Y-expressing tumors in mice
Jennifer A. Westwood,Mark J. Smyth,Michele W.L. Teng,Maria Moeller,Joseph A. Trapani,Andrew M. Scott,Fiona E Smyth,Glenn A Cartwright,Barbara E. Power,Dirk Hönemann,H. Miles Prince,Phillip K. Darcy,Michael H. Kershaw +12 more
TL;DR: It is demonstrated that anti-Le(Y) T cells preferentially expanded or accumulated in the tumor compared with control empty vector T cells, thereby providing mechanistic insight into the specific antitumor response.