B
Bayasi Guleng
Researcher at University of Tokyo
Publications - 21
Citations - 1102
Bayasi Guleng is an academic researcher from University of Tokyo. The author has contributed to research in topics: Gene knockdown & RNA interference. The author has an hindex of 11, co-authored 19 publications receiving 1067 citations.
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Journal ArticleDOI
Functional analysis of PIK3CA gene mutations in human colorectal cancer.
Tsuneo Ikenoue,Fumihiko Kanai,Yohko Hikiba,Toshiyuki Obata,Yasuo Tanaka,Jun Imamura,Miki Ohta,Amarsanaa Jazag,Bayasi Guleng,Keisuke Tateishi,Yoshinari Asaoka,Masayuki Matsumura,Takao Kawabe,Masao Omata +13 more
TL;DR: Results show that almost all the colon cancer-associated PIK3CA mutations are functionally active so that they are likely to be involved in carcinogenesis.
Journal ArticleDOI
Blockade of the Stromal Cell–Derived Factor-1/CXCR4 Axis Attenuates In vivo Tumor Growth by Inhibiting Angiogenesis in a Vascular Endothelial Growth Factor–Independent Manner
Bayasi Guleng,Keisuke Tateishi,Miki Ohta,Fumihiko Kanai,Amarsanaa Jazag,Hideaki Ijichi,Yasuo Tanaka,Miwa Washida,Keita Morikane,Yasushi Fukushima,Takao Yamori,Takashi Tsuruo,Takao Kawabe,Makoto Miyagishi,Kazunari Taira,Masataka Sata,Masao Omata +16 more
TL;DR: The data indicate that the SDF-1/CXCR4 pathway might be a general target for anticancer strategies and that blocking this system could be cooperatively effective in combination with other antiangiogenic therapies, such as blockade of VEGF.
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The hepatitis B virus X protein enhances AP-1 activation through interaction with Jab1.
Yasuo Tanaka,Fumihiko Kanai,Tohru Ichimura,Keisuke Tateishi,Yoshinari Asaoka,Bayasi Guleng,Amarsanaa Jazag,Miki Ohta,Jun Imamura,Tsuneo Ikenoue,Hideaki Ijichi,Takao Kawabe,Toshiaki Isobe,Masao Omata +13 more
TL;DR: Results suggest that the interaction between HBx and Jab1 enhances HBx-mediated AP-1 activation.
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p53-Independent Negative Regulation of p21/Cyclin-Dependent Kinase–Interacting Protein 1 by the Sonic Hedgehog-Glioma-Associated Oncogene 1 Pathway in Gastric Carcinoma Cells
Miki Ohta,Keisuke Tateishi,Fumihiko Kanai,Hirotsugu Watabe,Shintaro Kondo,Bayasi Guleng,Yasuo Tanaka,Yoshinari Asaoka,Amarsanaa Jazag,Jun Imamura,Hideaki Ijichi,Tsuneo Ikenoue,Masataka Sata,Makoto Miyagishi,Kazunari Taira,Minoru Tada,Takao Kawabe,Masao Omata +17 more
TL;DR: It is suggested that the overexpression of Shh contributes to constitutive Hh activation and that this signaling pathway negatively regulates p21/CIP1 through a Gli1-dependent and p53-independent mechanism in gastric carcinoma cells.
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Smad4 silencing in pancreatic cancer cell lines using stable RNA interference and gene expression profiles induced by transforming growth factor-beta.
Amarsanaa Jazag,Hideaki Ijichi,Fumihiko Kanai,Takaaki Imamura,Takaaki Imamura,Bayasi Guleng,Miki Ohta,Jun Imamura,Yasuo Tanaka,Keisuke Tateishi,Tsuneo Ikenoue,Takayuki Kawakami,Yoshihiro Arakawa,Makoto Miyagishi,Makoto Miyagishi,Kazunari Taira,Kazunari Taira,Takao Kawabe,Masao Omata +18 more
TL;DR: Genes that are involved in cell proliferation, adhesion, and motility were found to be regulated differentially with respect to S4KD and control cells, which suggests signaling pathways that differ according to Smad4 status.