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Bernhard A. Haug

Researcher at University of Göttingen

Publications -  11
Citations -  1336

Bernhard A. Haug is an academic researcher from University of Göttingen. The author has contributed to research in topics: Transcranial magnetic stimulation & Silent period. The author has an hindex of 9, co-authored 11 publications receiving 1281 citations.

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Axonal neuregulin-1 regulates myelin sheath thickness

TL;DR: This work uses mutant and transgenic mouse lines to show that axonal Neuregulin-1 (Nrg1) signals information about axon size to Schwann cells, and suggests a model by which myelin-forming SchwANN cells integrate axonal Nrg1 signals as a biochemical measure of axonsize.
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Silent period measurement revives as a valuable diagnostic tool with transcranial magnetic stimulation

TL;DR: A multivariate linear regression analysis revealed a positive correlation of SP with SI and a slight negative correlation with age, and in 11 hemiparetic patients a relative increase of the SP was found on the affected side despite normal central motor conduction time.
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Predominant affection of the blue cone pathway in Parkinson's disease.

TL;DR: It is concluded that the retinal deficit of dopamine in Parkinson's disease is reflected in diminished centre/surround inhibition and that these changes are primarily apparent when vision is tested along the tritan axis, because blue cones are sparsely distributed.
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Visual thresholds to low-contrast pattern displacement, color contrast, and luminance contrast stimuli in Parkinson's disease

TL;DR: The computerized Moorfield Vision System is used to demonstrate specific increases in various perceptual visual thresholds in idiopathic Parkinson's syndrome and concludes that the dopaminergic deficit of retinal amacrine cells in Parkinson patients can be monitored by combined low‐contrast and motion (displacement) stimuli.
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Latency and duration of the muscle silent period following transcranial magnetic stimulation in multiple sclerosis, cerebral ischemia, and other upper motoneuron lesions

TL;DR: In lesser degrees of paresis, SP duration is more sensitive than central motor conduction time (CMCT), but its specificity awaits further evaluation, and SP onset latency correlates well with CMCT and peripheral nerve conduction slowing, as in polyradiculoneuritis.