B
Bodil Nielsen
Researcher at University of Copenhagen
Publications - 69
Citations - 8174
Bodil Nielsen is an academic researcher from University of Copenhagen. The author has contributed to research in topics: Cerebral blood flow & Physical exercise. The author has an hindex of 42, co-authored 69 publications receiving 7753 citations.
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Journal ArticleDOI
Influence of body temperature on the development of fatigue during prolonged exercise in the heat
José González-Alonso,Christina Teller,Signe Lindgaard Andersen,Frank Jensen,Tino Hoffmann Hyldig,Bodil Nielsen +5 more
TL;DR: It is demonstrated that high internal body temperature per se causes fatigue in trained subjects during prolonged exercise in uncompensable hot environments and time to exhaustion in hot environments is inversely related to the initial temperature and directly related toThe rate of heat storage.
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Human circulatory and thermoregulatory adaptations with heat acclimation and exercise in a hot, dry environment.
TL;DR: It is concluded that the high core temperature per se, and not circulatory failure, is the critical factor for the exhaustion during exercise in heat stress.
Journal ArticleDOI
Hyperthermia and central fatigue during prolonged exercise in humans
Lars Nybo,Bodil Nielsen +1 more
TL;DR: The ability to generate force during a prolonged MVC is attenuated with hyperthermia, and the impaired performance is associated with a reduction in the voluntary activation percentage.
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Perceived exertion is associated with an altered brain activity during exercise with progressive hyperthermia
Lars Nybo,Bodil Nielsen +1 more
TL;DR: The linear correlation among core temperature, EEG frequency index, and RPE indicates that alterations in cerebral activity may be associated with the hyperthermia-induced development of fatigue during prolonged exercise in hot environments.
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Muscle blood flow is reduced with dehydration during prolonged exercise in humans
TL;DR: It is concluded that blood flow to the exercising muscles declines significantly with dehydration, due to a lowering in perfusion pressure and systemic blood flow rather than increased vasoconstriction, and the progressive increase in oxygen consumption during exercise is confined to the active muscles.