J
José González-Alonso
Researcher at Brunel University London
Publications - 122
Citations - 11528
José González-Alonso is an academic researcher from Brunel University London. The author has contributed to research in topics: Skeletal muscle & Hemodynamics. The author has an hindex of 54, co-authored 116 publications receiving 10548 citations. Previous affiliations of José González-Alonso include University of Copenhagen & University of Texas at Austin.
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Influence of body temperature on the development of fatigue during prolonged exercise in the heat
José González-Alonso,Christina Teller,Signe Lindgaard Andersen,Frank Jensen,Tino Hoffmann Hyldig,Bodil Nielsen +5 more
TL;DR: It is demonstrated that high internal body temperature per se causes fatigue in trained subjects during prolonged exercise in uncompensable hot environments and time to exhaustion in hot environments is inversely related to the initial temperature and directly related toThe rate of heat storage.
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Dehydration markedly impairs cardiovascular function in hyperthermic endurance athletes during exercise
TL;DR: It is demonstrated that the superimposition of dehydration on hyperthermia during exercise in the heat causes an inability to maintain cardiac output and blood pressure that makes the dehydrated athlete less able to cope withhyperthermia.
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Fluid and carbohydrate ingestion independently improve performance during 1 h of intense exercise.
TL;DR: Large FR slightly attenuates the increase in heart rate and core temperature which occurs during Small FR, and both fluid and carbohydrate ingestion equally improve cycling performance and their effects are additive.
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The cardiovascular challenge of exercising in the heat
TL;DR: This review focuses on how the cardiovascular system is regulated when exercising in the heat and how restrictions in locomotor skeletal muscle and/or skin perfusion might limit athletic performance in hot environments.
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Reductions in Systemic and Skeletal Muscle Blood Flow and Oxygen Delivery Limit Maximal Aerobic Capacity in Humans
TL;DR: These results demonstrate that in trained humans, severe heat stress reduces &OV0312;o2max by accelerating the declines in cardiac output and mean arterial pressure that lead to decrements in exercising muscle blood flow, O2 delivery, and O2 uptake.