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Brenda A. Wilson

Researcher at University of Illinois at Urbana–Champaign

Publications -  108
Citations -  8002

Brenda A. Wilson is an academic researcher from University of Illinois at Urbana–Champaign. The author has contributed to research in topics: Microbiome & Heterotrimeric G protein. The author has an hindex of 41, co-authored 105 publications receiving 6895 citations. Previous affiliations of Brenda A. Wilson include Harvard University & NHS Lothian.

Papers
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Journal ArticleDOI

Critical Evaluation of Two Primers Commonly Used for Amplification of Bacterial 16S rRNA Genes

TL;DR: This work proposes a formulation for a forward primer (27f) that includes three sequences not usually present that is better at maintaining the original rRNA gene ratio of Lactobacillus spp.
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Habitat degradation impacts black howler monkey ( Alouatta pigra ) gastrointestinal microbiomes

TL;DR: High throughput microbial 16S ribosomal RNA gene sequencing indicated that diversity, richness and composition of howler GI microbiomes varied with host habitat in relation to diet, and a reduction in the number of genes related to butyrate production and hydrogen metabolism in the microbiomes of howlers occupying suboptimal habitats may impact host health.
Book

Bacterial Pathogenesis : A Molecular Approach

TL;DR: Biosecurity: the Changing Roles of Microbiologists in an Age of Bioterrorism and Emerging Diseases is published, with a focus on the role of vaccine and the changing roles ofmicrobes and disease.
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Obesity during childhood and adolescence augments bone mass and bone dimensions

TL;DR: Obesity during childhood and adolescence was associated with increased vertebral bone density and increased whole-body bone dimensions and mass and these differences persisted after adjustment for obesity-related increases in height, maturation, and lean mass.
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Pasteurella multocida: from Zoonosis to Cellular Microbiology

TL;DR: Recent comparative genomics and molecular pathogenesis studies that have advanced the understanding of the multiple virulence mechanisms employed by Pasteurella species to establish acute and chronic infections are reviewed.