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Bruce N. Van Vliet

Researcher at Memorial University of Newfoundland

Publications -  45
Citations -  1246

Bruce N. Van Vliet is an academic researcher from Memorial University of Newfoundland. The author has contributed to research in topics: Blood pressure & Baroreflex. The author has an hindex of 19, co-authored 45 publications receiving 1173 citations. Previous affiliations of Bruce N. Van Vliet include St. John's University.

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Increases in CSF [Na+] precede the increases in blood pressure in Dahl S rats and SHR on a high-salt diet.

TL;DR: In Dahl salt-sensitive and salt-resistant rats, and spontaneously hypertensive rats (SHR) and Wistar-Kyoto rats, at 5-6 wk of age, a cannula was placed in the cisterna magna, and cerebrospinal fluid (CSF) was withdrawn continuously, demonstrating a primary role of increased CSF [Na+] in the salt-induced hypertension.
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Direct and indirect methods used to study arterial blood pressure

TL;DR: Telemeter implantation methods in mice are presently difficult, with relatively low success rates being reported, but validation of new methods, such as the insertion of the catheter tip via the carotid artery, may make the technique more widely accessible in the near future.
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Characteristics of 24 h Telemetered Blood Pressure in eNOS‐Knockout and C57Bl/6J Control Mice

TL;DR: The results indicate that eNOS−/− mice have mild hypertension that is accompanied by more pronounced increases in BP lability and/or reactivity and also demonstrate a marked effect of locomotor activity on BP in mice, which may confound short‐term measurements of BP.
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Understanding the contribution of Guyton's large circulatory model to long‐term control of arterial pressure

TL;DR: Guyton's circulatory model was particularly instrumental in exploring the linkage between blood pressure and sodium balance and in demonstrating an overriding importance of renal salt and water balance in setting the long‐term blood pressure level.
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Role of mutation of the circadian clock gene Per2 in cardiovascular circadian rhythms

TL;DR: Data show that mutation of the Period2 gene results in an attenuated dipping of blood pressure and heart rate during both light-dark cycles and constant darkness, and in shorter circadian periods during constant darkness.