C
Carl W. Schmid
Researcher at University of California, Davis
Publications - 102
Citations - 8705
Carl W. Schmid is an academic researcher from University of California, Davis. The author has contributed to research in topics: Alu element & Gene. The author has an hindex of 48, co-authored 102 publications receiving 8524 citations. Previous affiliations of Carl W. Schmid include University of California, Berkeley.
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Evolution of alu family repeats since the divergence of human and chimpanzee
Ikuhisa Sawada,Cary Willard,Che Kun James Shen,Barbara Chapman,Allan C. Wilson,Carl W. Schmid +5 more
TL;DR: The sequences of seven pairs of chimpanzee and human Alu repeats are compared, finding that the identical Alu repeat is located at identical sites in the human and chimpanzee genomes.
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Isolation and chromosomal localization of highly repeated DNA sequences in Drosophila melanogaster.
TL;DR: The nuclear DNA of D. melanogaster contains DNA sequences that are repeated between ten and a hundred times more often than the next class of redundant DNA, which has been localized by "in situ" hybridization in the chromocenter of the chromosomes of the salivary gland.
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Specific Alu binding protein from human sperm chromatin prevents DNA methylation.
Igor Chesnokov,Carl W. Schmid +1 more
TL;DR: This sperm Alu binding protein selectively protects Alu elements from methylation in vitro and may be responsible for the unmethylated state of Alu sequences in the male germ line resulting in a parent-specific differential inheritance of AlU methylation.
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Genome-wide chromatin remodeling modulates the Alu heat shock response.
TL;DR: During heat shock recovery in Hela cells, the level of Alu RNA transiently increases with kinetics that approximately parallel the transient expression of heat shock protein mRNAs suggesting that an opening and re-closing of chromatin regulates the Alu stress response.
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Silk worm Bm1 SINE RNA increases following cellular insults.
TL;DR: Results support the proposal that Sine RNAs serve a role in the cell stress response that predates the divergence of insects and mammals implying that SINEs are essentially a class of cell stress genes.