Chenglian Bai

TL;DR: This is the first study to define uptake kinetics and to focus on behavioral consequences following PFOS exposure in zebrafish, and further the understanding of the toxicity of PFOS to aquatic organisms and suggest the need for additional research to identify the mode ofPFOS toxicity.

TL;DR: It is demonstrated that the altered patterns of neuronal connectivity may contribute to motor behavior deficits, indicating the relevance of zebrafish as a model for studying toxicant developmental neurotoxicity.

TL;DR: It is revealed that parental chronic low dose BDE-209 exposure not only affects F0 growth and reproduction, but also elicits neurobehavior alternations in F1 offspring.

TL;DR: The results suggested that low dose of developmental lead exposure resulted in embryonic toxicity, behavioral alteration, and adult learning/memory deficit in zebrafish.

TL;DR: Zebrafish embryo is utilized as a model system to investigate the effect of developmental BPA exposure on larval teratology, motor behaviors, axonal growth of spinal motoneurons and muscle structure at various developmental stages, suggesting the functional relevance of muscle structural changes and the observed deficits in swimming activity.