C
Christoph F. Batzilla
Researcher at University of Würzburg
Publications - 4
Citations - 407
Christoph F. Batzilla is an academic researcher from University of Würzburg. The author has contributed to research in topics: Staphylococcus epidermidis & Mutant. The author has an hindex of 4, co-authored 4 publications receiving 392 citations.
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Journal ArticleDOI
Nosocomial infections by Staphylococcus epidermidis: how a commensal bacterium turns into a pathogen.
Wilma Ziebuhr,Susanne Hennig,Martin Eckart,Hennes Kränzler,Christoph F. Batzilla,Svetlana Kozitskaya +5 more
TL;DR: In this paper, epidemiological analyses using multilocus sequence typing (MLST) and genetic studies suggest that S. epidermidis isolates in the hospital environment differ from those obtained outside of medical facilities with respect to biofilm formation, antibiotic resistance, and the presence of mobile DNA elements.
Journal ArticleDOI
Proteomic analysis of Legionella-containing phagosomes isolated from Dictyostelium.
Olga Shevchuk,Christoph F. Batzilla,Sonja Hägele,Harald Kusch,Susanne Engelmann,Michael Hecker,Albert Haas,Klaus Heuner,Gernot Glöckner,Michael Steinert +9 more
TL;DR: A kinetic dissection ofphagosome maturation including the complex alterations of the phagosome protein composition is presented, including the putative cysteine proteinase inhibitor interference with lysosomal enzyme sorting and activation processes.
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Impact of the accessory gene regulatory system (Agr) on extracellular proteins, codY expression and amino acid metabolism in Staphylococcus epidermidis.
Christoph F. Batzilla,Shwan Rachid,Susanne Engelmann,Michael Hecker,Jörg Hacker,Wilma Ziebuhr +5 more
TL;DR: The combined data show that wild‐type and agr mutant differ with respect to amino acid biosynthesis and oligopeptide transport, carbohydrate utilization, as well as GMP and IMP interconversion, and might be capable of colonizing alternative ecological niches in the human host and have an advantage in adapting to changing environmental conditions.
Journal ArticleDOI
Staphylococcus aureus ClpC ATPase is a late growth phase effector of metabolism and persistence
Indranil Chatterjee,Sigrid Schmitt,Christoph F. Batzilla,Susanne Engelmann,Andreas Keller,Michael W. Ring,Ralf Kautenburger,Wilma Ziebuhr,Michael Hecker,Klaus T. Preissner,Markus Bischoff,Richard A. Proctor,Horst P. Beck,Hans-Peter Lenhof,Greg A. Somerville,Mathias Herrmann +15 more
TL;DR: This study provides strong evidence for ClpC as a critical factor in staphylococcal energy metabolism, stress regulation, and late‐stationary phase survival; therefore, these data provide important insight into the adaptation of S. aureus toward a persister state in chronic infections.