D
Dae Ho Cho
Researcher at Sookmyung Women's University
Publications - 53
Citations - 2133
Dae Ho Cho is an academic researcher from Sookmyung Women's University. The author has contributed to research in topics: MAPK/ERK pathway & Apoptosis. The author has an hindex of 27, co-authored 53 publications receiving 1821 citations.
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Air pollution and skin diseases: Adverse effects of airborne particulate matter on various skin diseases
TL;DR: Overall, increased PM levels are highly associated with the development of various skin diseases via the regulation of oxidative stress and inflammatory cytokines, therefore, anti-oxidant and anti-inflammatory drugs may be useful for treating PM-induced skin diseases.
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Adiponectin is a negative regulator of NK cell cytotoxicity
Kun-yong Kim,Jae Kwang Kim,Seung Hyun Han,Jong-Seok Lim,Keun Il Kim,Dae Ho Cho,Myeong Sok Lee,Jeong Hyung Lee,Do Young Yoon,Suk Ran Yoon,Jin Woong Chung,Inpyo Choi,Eunjoon Kim,Young Yang +13 more
TL;DR: Results clearly demonstrate that adiponectin is a potent negative regulator of IL-2-induced NK cell activation and thus may act as an in vivo regulator of anti-inflammatory functions.
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Rosacea: Molecular Mechanisms and Management of a Chronic Cutaneous Inflammatory Condition
TL;DR: The genetic predisposition for rosacea along with its associated diseases, triggering factors, and suggested management options are described in detail based on the underlying molecular biology.
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l-Kynurenine-induced apoptosis in human NK cells is mediated by reactive oxygen species
Hyunkeun Song,Hyun Jin Park,Yeong Seok Kim,Kwang Dong Kim,Hyun Kyung Lee,Dae Ho Cho,Jae Wook Yang,Dae Young Hur +7 more
TL;DR: It is concluded that L-kynurenine resulting from IDO can cause cell death via ROS pathway in NK cells, providing a new insight into the interaction between NK cells and IDO positive cancer cells in regulating immune responses.
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Molecular mechanisms of cutaneous inflammatory disorder: Atopic dermatitis
TL;DR: A review of the genetics, epigenetics, barrier dysfunction and immunological abnormalities in AD with a focus on updated molecular biology is presented.