D
David B. Collinge
Researcher at University of Copenhagen
Publications - 110
Citations - 9182
David B. Collinge is an academic researcher from University of Copenhagen. The author has contributed to research in topics: Hordeum vulgare & Powdery mildew. The author has an hindex of 41, co-authored 100 publications receiving 8028 citations. Previous affiliations of David B. Collinge include University of Copenhagen Faculty of Life Sciences.
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Journal ArticleDOI
Subcellular localization of H2O2 in plants. H2O2 accumulation in papillae and hypersensitive response during the barley—powdery mildew interaction
TL;DR: 3,3-diaminobenzidine polymerizes instantly and locally as soon as it comes into contact with H2O2 in the presence of peroxidase, and it was found that, by allowing the leaf to take up this substrate, in-vivo and in-situ detection of H2 O2 can be made at subcellular levels.
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Plant chitinases
David B. Collinge,Karsten Matthias Kragh,Jørn Dalgaard Mikkelsen,Klaus K. Nielsen,Ulla Rasmussen,Knud Vad +5 more
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Roles of reactive oxygen species in interactions between plants and pathogens
Nandini P. Shetty,Hans Jørgen Lyngs Jørgensen,J. D. Jensen,David B. Collinge,H. Shekar Shetty +4 more
TL;DR: This review will assess the different roles of ROS in host–pathogen interactions with special emphasis on fungal and Oomycete pathogens.
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Germin-like oxalate oxidase, a H2O2-producing enzyme, accumulates in barley attacked by the powdery mildew fungus
TL;DR: Increased activity of this enzyme in association with the response of barley to the powdery mildew fungus, Erysiphe graminis f.sp.
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Role of hydrogen peroxide during the interaction between the hemibiotrophic fungal pathogen Septoria tritici and wheat.
Nandini P. Shetty,Rahim Mehrabi,Henrik Lütken,Anna Haldrup,Gert H. J. Kema,David B. Collinge,Hans Jørgen Lyngs Jørgensen +6 more
TL;DR: Infection by necrotrophs or water into wheat during the biotrophic or the necrotrophic phase of S. tritici and the effect of infection on host physiology is studied to get an understanding of the survival strategy of the pathogen.