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David C. Good

Researcher at Pennsylvania State University

Publications -  50
Citations -  2287

David C. Good is an academic researcher from Pennsylvania State University. The author has contributed to research in topics: Stroke & Working memory. The author has an hindex of 23, co-authored 48 publications receiving 2042 citations. Previous affiliations of David C. Good include Wake Forest University & Penn State Milton S. Hershey Medical Center.

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Sleep-Disordered Breathing and Poor Functional Outcome After Stroke

TL;DR: SDB accompanied by arterial oxyhemoglobin desaturation is common in patients undergoing rehabilitation after stroke and is associated with higher mortality at 1 year and lower BI scores at discharge and at 3 and 12 months after stroke.
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Constraint-Induced Movement Therapy Results in Increased Motor Map Area in Subjects 3 to 9 Months After Stroke

TL;DR: This first multi-center effort to measure cortical reorganization induced by CIMT in subjects who are in the subacute stage of recovery produced statistically significant and clinically relevant improvements in arm motor function that persisted for at least 4 months.
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Dose-dependent response to intramuscular botulinum toxin type A for upper-limb spasticity in patients after a stroke.

TL;DR: Childers et al. as mentioned in this paper showed that intramuscular botulinum toxin type A (BTX) reduces excessive muscle tone in a dose-dependent manner in the elbow, wrist, and fingers of patients who experience spasticity after a stroke.
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The Rich Get Richer: Brain Injury Elicits Hyperconnectivity in Core Subnetworks

TL;DR: The current findings generally support the hyperconnectivity hypothesis showing that during the first year of recovery after TBI, neural networks show increased connectivity, and this change is disproportionately represented in brain regions belonging to the brain's core subnetworks.
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Contralesional motor deficits after unilateral stroke reflect hemisphere-specific control mechanisms

TL;DR: The results provide the first demonstration of hemisphere specific motor control deficits in the contralesional arm of stroke patients and suggest that it is critical to consider the differential deficits induced by right or left hemisphere lesions to enhance post-stroke rehabilitation interventions.