David M. Jacobowitz

TL;DR: The evolutionarily conserved calcium binding domains and connecting regions, in addition to the limited local changes observed between rat and chick primary structure, lead us to believe that calretinin interacts with other highly conserved constituents of brain cells.

TL;DR: Results indicate that the expression of these related calcium‐binding proteins is altered in the GEPRs before the induction of seizures, which could alter either the calcium‐buffering capacity or regulation of calcium‐mediated processes by these proteins and thus play a role in the molecular cascade of events inducing the genetic susceptibility to, and the generalization of, seizures in these rat strains.

TL;DR: It is suggested that retinal deafferentation results in an increase in contents of calretinin in some cell bodies within the upper layers of the superior colliculus.

TL;DR: In contrast to the increase in heart rate observed following α-MSH injection into the dorsomedial nucleus, injections into the medial preoptic, anterior, paraventricular or posterior hypothalamic nuclei had no significant effects on blood pressure and heart rate.

TL;DR: Trigeminal primary neuronal cell bodies were labeled by retrograde transport of Fluoro-gold from the nasal mucosa of rats and processed for double stain for calretinin- and tachykinin-immunoreactivities (CR- and TK-irs).