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David M. Jacobowitz

Researcher at National Institutes of Health

Publications -  389
Citations -  26961

David M. Jacobowitz is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Calretinin & Hypothalamus. The author has an hindex of 84, co-authored 389 publications receiving 26673 citations. Previous affiliations of David M. Jacobowitz include Uniformed Services University of the Health Sciences & Hebrew University of Jerusalem.

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Selective action of 6-hydroxydopa on noradrenergic terminals: Mapping of preterminal axons of the brain

TL;DR: It is suggested that 6-hydroxydopa has the ability to cause selective destruction and/or marked impairment of noradrenergic terminals, which in the brainstem, are capable of regeneration or replenishment of the neurotransmitter.
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A histochemical study of the adrenergic and cholinergic innervation of the anterior segment of the rabbit eye.

TL;DR: The innervation of the anterior segment of the albino rabbit eye was studied with both the histofluorometric technique of Falck for adrenergic fibers and the thiocholine method for cholinergic fibers to find fibers found in heavy concentration in the iris dilator, ciliary processes, and around blood vessels.
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Distribution of corticotropin-releasing factor receptors in primate brain.

TL;DR: The data demonstrate that receptors for CRF are present within the primate brain at areas related to the central control of visceral function and behavior, suggesting that brain CRF may serve as a neurotransmitter in the coordination of endocrine and neural mechanisms involved in the response to stress.
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Effect of dexamethasone on phenylethanolamine n-methyltransferase in chromaffin tissue of the neonatal rat

TL;DR: Treatment of neonatal rats with dexamethasone resulted in the appearance of phenylethanolamine‐N‐methyltransferase (PNMT) and numerous small, intensely fluorescent (SIF) cells in abdominal paraganglia and in sympathetic paravertebral ganglia, which may be derived from a primitive stem cell precursor.
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Effects of estrogen and progesterone on plasma gonadotropins and on catecholamine levels and turnover in discrete brain regions of ovariectomized rats.

TL;DR: The results suggest that catecholamine neurons in several discrete brain regions participate in the stimulatory and inhibitory feedback effects of ovarian hormones on gonadotropin secretion, and perhaps also on the hormonal induction of sexual receptivity.