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David M. Jacobowitz

Researcher at National Institutes of Health

Publications -  389
Citations -  26961

David M. Jacobowitz is an academic researcher from National Institutes of Health. The author has contributed to research in topics: Calretinin & Hypothalamus. The author has an hindex of 84, co-authored 389 publications receiving 26673 citations. Previous affiliations of David M. Jacobowitz include Uniformed Services University of the Health Sciences & Hebrew University of Jerusalem.

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Calretinin-immunoreactivity in the oro-facial and pharyngeal regions of the rat.

TL;DR: It is indicated that viscerosensory (probably including gustatory) nerve fibers innervating the oral and pharyngeal tissues contain CR, while somotosensory nerve fiber innervates the facial skin are devoid of CR.
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Proteins Regulated by Gonadal Steroids in the Medial Preoptic and Ventromedial Hypothalamic Nuclei of Male and Female Rats

TL;DR: Protein profiles of brain areas mediating effects of steroid hormones on copulation were compared between animals in gonadal steroid states predictive of either the presence or absence of copulatory activity and several interesting reversal patterns were noted.
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Lack of biochemical evidence for a direct habenulo-raphe GABAergic pathway.

TL;DR: The results of this study do not support the hypothesis that a direct GABAergic pathway exists between the LHb and the dr nuclei, and the activity of GAD in the dr was measured in rats with habenular lesions following different survival periods.
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Calbindin-D28k-immunoreactivity in the trigeminal ganglion neurons and molar tooth pulp of the rat.

TL;DR: The present study indicated that the tooth pulp primary neurons contained CB-ir but did not coexpress CB- and CR-irs and that these neurons projected their myelinated axons to the pulp.
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Gender dependence for a subset of the low-abundance signaling proteome in human platelets.

TL;DR: It is reported here that platelets from male donors express significantly higher levels of signaling cascade proteins than platelet from female donors, which may suggest a biological mechanism for gender discrimination in cardiovascular disease.