D
David O. Beenhouwer
Researcher at University of California, Los Angeles
Publications - 47
Citations - 2578
David O. Beenhouwer is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Veterans Affairs & Cryptococcus neoformans. The author has an hindex of 16, co-authored 43 publications receiving 2321 citations. Previous affiliations of David O. Beenhouwer include University of California, Berkeley & United States Department of Veterans Affairs.
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Journal ArticleDOI
Granulomatous Infectious Diseases Associated with Tumor Necrosis Factor Antagonists
TL;DR: The clustering of reports shortly after initiation of treatment with infliximab is consistent with reactivation of latent infection, and a risk of granulomatous infection was 3.25-fold greater among patients who received inflIXimab than among those who received etanercept.
Journal ArticleDOI
Production of human monoclonal antibody in eggs of chimeric chickens
Lei Zhu,Marie-Cecile van de Lavoir,Jenny Albanese,David O. Beenhouwer,Pina M. Cardarelli,Severino Cuison,David F Deng,Shrikant Deshpande,Jennifer H. Diamond,Lynae Green,Edward L. Halk,Babette S. Heyer,Robert M. Kay,Allyn Kerchner,Philip Albert Leighton,Christine Mather,Sherie L. Morrison,Zivko L. Nikolov,David Passmore,Alicia Pradas-Monne,Benjamin T. Preston,Vangipuram S. Rangan,Mingxia Shi,Mohan Srinivasan,Steven G. White,Peggy Winters-Digiacinto,Susan Wong,Wen Zhou,Robert J. Etches +28 more
TL;DR: The ability of upstream and downstream DNA sequences of ovalbumin, a protein produced exclusively in very high quantities in chicken egg white, to drive tissue-specific expression of human mAb in chicken eggs is examined.
Journal ArticleDOI
Tumor Necrosis Factor Antagonists: Different Kinetics and/or Mechanisms of Action May Explain Differences in the Risk for Developing Granulomatous Infection
TL;DR: There are significant differences between the 2 classes of TNF antagonists in terms of both their kinetics and mechanisms of action, which may help explain the apparent differences in the incidence of granuloma-dependent infections among them.
Journal ArticleDOI
Granulomatous Infections Due to Tumor Necrosis Factor Blockade: Correction
Journal ArticleDOI
Staphylococcus aureus Panton-Valentine Leukocidin Contributes to Inflammation and Muscle Tissue Injury
Ching Wen Tseng,Ching Wen Tseng,Pierre Kyme,Pierre Kyme,Jennifer E Low,Jennifer E Low,Miguel A. Rocha,Miguel A. Rocha,Randa Alsabeh,Loren G. Miller,Michael Otto,Moshe Arditi,Moshe Arditi,Binh An Diep,Victor Nizet,Terence M. Doherty,Terence M. Doherty,David O. Beenhouwer,David O. Beenhouwer,George Y. Liu,George Y. Liu +20 more
TL;DR: In a model of necrotizing soft tissue infection, PVL caused significant damage of muscle but not the skin, and the toxin could mediate tissue injury by mechanisms other than direct killing of phagocytes.