Deborah A. Ross

TL;DR: It is indicated that GPX1 is the major, if not the only, metabolic form of body Se that protects mice against the lethal oxidative stress caused by high levels of paraquat; it seems less important, however, in protecting dogs against the moderate oxidative stress by the low level of paraaquat.

TL;DR: Results indicate that GPX1 is expressed independently of GPX3 or GPX4 and represents approximately 60% of the total hepatic Se in Se-adequate mice.

TL;DR: During insulin clamp treatments, the mammary gland was able to support the increased milk protein yields by increasing extraction efficiency of essential amino acids, mammary blood flow, and glucose uptake, and a positive mammary balance of total amino nitrogen and carbon was maintained for all treatments.

TL;DR: GPX1 is the major selenoprotein to protect mice against the lethal oxidative stress induced by diquat, and Responses of hepatic superoxide dismutase activities to the diqu at treatment were affected by the GPX1 level.

TL;DR: The microbiota in the ileum of untreated mice differed greatly from that of the cecum of the same mice, primarily among families of the phylum Firmicutes, and the pathology of Salmonella infection in the iileum was characterized.