E
E. Carboni
Researcher at University of Cagliari
Publications - 20
Citations - 4340
E. Carboni is an academic researcher from University of Cagliari. The author has contributed to research in topics: Nucleus accumbens & Dopamine. The author has an hindex of 17, co-authored 17 publications receiving 4209 citations.
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Journal ArticleDOI
Dopamine and Drug Addiction: The Nucleus Accumbens Shell Connection
Gaetano Di Chiara,Valentina Bassareo,Sandro Fenu,Maria Antonietta De Luca,Liliana Spina,C Cadoni,Elio Maria Gioachino Acquas,E. Carboni,Valentina Valentini,Daniele Lecca +9 more
TL;DR: It is speculated that drug addiction results from the impact exerted on behavior by the abnormal DA stimulant properties acquired by drug-conditioned stimuli as a result of their association with addictive drugs.
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Amphetamine, cocaine, phencyclidine and nomifensine increase extracellular dopamine concentrations preferentially in the nucleus accumbens of freely moving rats.
TL;DR: In contrast with amphetamine, cocaine, phencyclidine and nomifensine increase synaptic dopamine concentrations in vivo by a mechanism which depends on intact activity of dopaminergic neurons and by an exocytotic process, which is insensitive to these manipulations.
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Cocaine and Amphetamine Increase Extracellular Dopamine in the Nucleus Accumbens of Mice Lacking the Dopamine Transporter Gene
E. Carboni,Cécile Spielewoy,Cinzia Vacca,Marika Nosten-Bertrand,Bruno Giros,Gaetano Di Chiara +5 more
TL;DR: It is reported that, in DAT-KO mice, cocaine and amphetamine increase dialysate dopamine in the medial part of the nucleus accumbens, supporting the hypothesis of a primary role of nucleus accumens dopamine in drug reinforcement.
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Increase of extracellular dopamine in the prefrontal cortex: a trait of drugs with antidepressant potential?
TL;DR: The possibility that the property of stimulating dopamine transmission in the prefrontal cortex has a role in the antidepressant properties of these drugs is raised.
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Differential inhibitory effects of a 5-HT3 antagonist on drug-induced stimulation of dopamine release.
TL;DR: The results indicate that blockade of 5-HT3 receptors selectively prevents the stimulation of DA release induced by drugs known to stimulate the firing activity of DA neurons.