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F. Dallemer

Researcher at Centre national de la recherche scientifique

Publications -  17
Citations -  245

F. Dallemer is an academic researcher from Centre national de la recherche scientifique. The author has contributed to research in topics: Axial chirality & Phosphole. The author has an hindex of 9, co-authored 17 publications receiving 206 citations. Previous affiliations of F. Dallemer include Rhône-Poulenc & Rhodia.

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Can geometry control the coordination behaviour of 2-hydroxy-1-naphthaldehyde-N(4)-phenylthiosemicarbazone? A study towards its origin

TL;DR: In this paper, the 2-hydroxy-1-naphthaldehyde-N(4)-phenylthiosemicarbazone (H 2 -Nap-ptsc) was reacted with square planar complexes of the type [MCl 2 (EPh 3 ) 2 ] (where M = Ni or Pd; E = P or As) and octahedral [RuHCl(CO)(PPh 3 ] 3 ].
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CT-DNA/BSA protein binding and antioxidant studies of new binuclear Pd(II) complexes and their structural characterisation

TL;DR: In this paper, the true coordinating nature of the ligands in the binuclear complexes was confirmed by X-ray crystallographic studies, and electrostatic and intercalative binding modes have been proposed.
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Organoruthenium(II) Complexes Ameliorates Oxidative Stress and Impedes the Age Associated Deterioration in Caenorhabditis elegans through JNK-1/DAF-16 Signalling.

TL;DR: It is found that new ruthenium(II) complexes 1–4 confered tolerance to oxidative stress and extend the mean lifespan of mev-1 mutant worms and wild-type Caenorhabditis elegans and it is hoped that new Ru(ƞ6-p-cymene) complexes could be further considered as a potential drug to retard aging and age-related neurodegenerative diseases.
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Organoruthenium(II) complexes attenuate stress in Caenorhabditis elegans through regulating antioxidant machinery.

TL;DR: The genetic and reporter gene expression analysis revealed that complexes 2-4 maintained the intracellular redox status and offered stress protection through transactivation of antioxidant defence machinery genes gst-4 and sod-3 which are directly regulated by SKN-1 and DAF-16 transcription factors, respectively.