F
Fabio Galetta
Researcher at University of Pisa
Publications - 143
Citations - 4675
Fabio Galetta is an academic researcher from University of Pisa. The author has contributed to research in topics: Blood pressure & Heart rate. The author has an hindex of 37, co-authored 143 publications receiving 4336 citations.
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Journal ArticleDOI
Physical activity prevents age-related impairment in nitric oxide availability in elderly athletes.
Stefano Taddei,Fabio Galetta,Agostino Virdis,Lorenzo Ghiadoni,Guido Salvetti,Ferdinando Franzoni,Costantino Giusti,Antonio Salvetti +7 more
TL;DR: It is suggested that regular physical activity can at least in part prevent the age-induced endothelial dysfunction, probably the restoration of nitric oxide availability consequent to prevention of production of oxidative stress.
Journal ArticleDOI
Physical activity, plasma antioxidant capacity, and endothelium-dependent vasodilation in young and older men
Ferdinando Franzoni,Lorenzo Ghiadoni,Fabio Galetta,Y. Plantinga,Valter Lubrano,Yale Huang,Guido Salvetti,Francesco Regoli,Stefano Taddei,Gino Santoro,Antonio Salvetti +10 more
TL;DR: The results suggest that regular physical activity is associated with preserved antioxidant defenses and endothelial function in older individuals.
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A comparative study of the in vitro antioxidant activity of statins
TL;DR: All the statins tested have intrinsic antioxidant activity with both anti-hydroxyl and peroxyl radical activity, and Simvastatin was the most effective as an anti-Hydroxyl radical antioxidant and fluvastsatin as anAnti-peroxylradical antioxidant.
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Heart rate variability and left ventricular diastolic function in anorexia nervosa
TL;DR: The findings demonstrate a cardiovascular vagal hyperactivity in AN, which appears to influence the ventricular diastolic dynamics, and HRV and diastolics function analysis may represent useful tools in monitoring anorexia-induced cardiac modifications.
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Mechanisms of Coronary Flow Reserve Impairment in Human Hypertension An Integrated Approach by Transthoracic and Transesophageal Echocardiography
TL;DR: It is confirmed that coronary reserve in hypertensive individuals is reduced even before the occurrence of left ventricular hypertrophy, and the reduction in coronary reserve depends on both an increase in resting coronary flow and an impairment in maximal vasodilator capacity.