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Fang Liu

Researcher at University of Georgia

Publications -  43
Citations -  1744

Fang Liu is an academic researcher from University of Georgia. The author has contributed to research in topics: Subventricular zone & Neuroblast. The author has an hindex of 19, co-authored 40 publications receiving 1463 citations. Previous affiliations of Fang Liu include Fudan University & BioMérieux.

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Identification and characterization of neuroblasts in the subventricular zone and rostral migratory stream of the adult human brain

TL;DR: Evidence that neuroblasts exist continuously in the anterior ventral SVZ and RMS of the adult human brain is provided and the data suggest that the SVZ maintains the ability to produce neuroblast in the adulthuman brain.
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Subcortical origins of human and monkey neocortical interneurons

TL;DR: It is proposed that the majority of primate neocortical GABAergic interneurons originate from ganglionic eminences of the ventral telencephalon, and this work reveals that the mammalian neocortex shares basic rules for interneuron development, substantially reshaping the understanding of the origin and classification ofPrimate neocortex.
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Identification and Validation of Long Noncoding RNA Biomarkers in Human Non–Small-Cell Lung Carcinomas

TL;DR: Multiple novel lncRNAs associated with tumorigenesis and histological differentiation in human NSCLC have been identified and validated and could be further exploited for the development of useful biomarkers in diagnosis, prognosis, and treatment ofNSCLC.
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Brain Injury Does Not Alter the Intrinsic Differentiation Potential of Adult Neuroblasts

TL;DR: It is shown that SVZ neuroblasts give rise almost exclusively to calretinin-expressing cells in the damaged striatum, resulting in the accumulation of these cells during long term recovery after stroke, and suggests that adult neuroblast do not alter their intrinsic differentiation potential after brain injury.
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Lrp4 in astrocytes modulates glutamatergic transmission

TL;DR: A critical role is revealed for Lrp4, in response to agrin, in modulating astrocytic ATP release and synaptic transmission in mice lacking low-density lipoprotein receptor–related protein 4, a protein that is critical for neuromuscular junction formation.